Full Transcript Early Cognitive Decline and Misdiagnosis 0:03 two years. For two years, she told her 0:03 two years. For two years, she told her doctors that something was happening to 0:05 doctors that something was happening to 0:05 doctors that something was happening to her brain. Words that just didn't come 0:08 her brain. Words that just didn't come 0:08 her brain. Words that just didn't come as fast. A fog that sleep just didn't 0:11 as fast. A fog that sleep just didn't 0:11 as fast. A fog that sleep just didn't fix. A version of herself she just 0:14 fix. A version of herself she just 0:14 fix. A version of herself she just couldn't quite get back to. Two doctors, 0:17 couldn't quite get back to. Two doctors, 0:17 couldn't quite get back to. Two doctors, two years, and what she was told 0:20 two years, and what she was told 0:20 two years, and what she was told consistently was, "It's just stress. 0:23 consistently was, "It's just stress. 0:23 consistently was, "It's just stress. This is just pmenopause. This is just 0:25 This is just pmenopause. This is just 0:25 This is just pmenopause. This is just normal aging." When she came to me, I
Introducing the Patient Case and Diagnostic Approach 0:28 normal aging." When she came to me, I 0:28 normal aging." When she came to me, I ran the markers that most physicians 0:30 ran the markers that most physicians 0:30 ran the markers that most physicians never order. And what we found changed 0:34 never order. And what we found changed 0:34 never order. And what we found changed the trajectory of her next decade. Just 0:37 the trajectory of her next decade. Just 0:37 the trajectory of her next decade. Just aging is not a diagnosis. And the 0:40 aging is not a diagnosis. And the 0:40 aging is not a diagnosis. And the cognitive decline, which is real, 0:43 cognitive decline, which is real, 0:43 cognitive decline, which is real, measurable, reversible in many cases, 0:46 measurable, reversible in many cases, 0:46 measurable, reversible in many cases, begins years before it even has a name. 0:49 begins years before it even has a name. 0:49 begins years before it even has a name. I'm Dr. Myra Reid, and I practice early 0:52 I'm Dr. Myra Reid, and I practice early 0:52 I'm Dr. Myra Reid, and I practice early detection longevity medicine. My
Why Cognitive Decline Is Not Inevitable 0:54 detection longevity medicine. My 0:54 detection longevity medicine. My practice runs through four pillars: 0:57 practice runs through four pillars: 0:57 practice runs through four pillars: hormones, gut health, lifestyle factors, 1:01 hormones, gut health, lifestyle factors, 1:01 hormones, gut health, lifestyle factors, and environmental exposures. I'd like to 1:04 and environmental exposures. I'd like to 1:04 and environmental exposures. I'd like to tell you about a patient, and for her 1:06 tell you about a patient, and for her 1:06 tell you about a patient, and for her name's privacy, we'll call her Carol. 1:08 name's privacy, we'll call her Carol. 1:08 name's privacy, we'll call her Carol. Carol was 61 years old when she started 1:10 Carol was 61 years old when she started 1:10 Carol was 61 years old when she started coming to see me. She had been a sharp, 1:13 coming to see me. She had been a sharp, 1:13 coming to see me. She had been a sharp, high functioning professional her entire 1:15 high functioning professional her entire 1:15 high functioning professional her entire career. And in the two years before she 1:17 career. And in the two years before she 1:17 career. And in the two years before she found me, she had been slowly watching 1:20 found me, she had been slowly watching 1:20 found me, she had been slowly watching that version of herself kind of 1:22 that version of herself kind of 1:22 that version of herself kind of disappear. She was losing words about 1:25 disappear. She was losing words about 1:25 disappear. She was losing words about mids sentence, not occasionally but 1:27 mids sentence, not occasionally but 1:27 mids sentence, not occasionally but consistently. And she would walk into a 1:30 consistently. And she would walk into a 1:30 consistently. And she would walk into a room and had no idea why she was there. 1:33 room and had no idea why she was there. 1:33 room and had no idea why she was there. She would read the same paragraph three 1:35 She would read the same paragraph three
Key Upstream Drivers of Brain Health 1:36 She would read the same paragraph three times and retain nothing. And she would 1:38 times and retain nothing. And she would 1:38 times and retain nothing. And she would wake up at 3:00 a.m. unable to fall back 1:41 wake up at 3:00 a.m. unable to fall back 1:41 wake up at 3:00 a.m. unable to fall back asleep. She told her primary care doctor 1:44 asleep. She told her primary care doctor 1:44 asleep. She told her primary care doctor and she was told it was just stress. She 1:46 and she was told it was just stress. She 1:46 and she was told it was just stress. She went to a specialist and she was told it 1:48 went to a specialist and she was told it 1:48 went to a specialist and she was told it was pmenopause. 1:50 was pmenopause. 1:50 was pmenopause. Both times she was handed a prescription 1:53 Both times she was handed a prescription 1:53 Both times she was handed a prescription and sent home. No cognitive testing 1:56 and sent home. No cognitive testing 1:56 and sent home. No cognitive testing done, no metabolic workup, no 1:58 done, no metabolic workup, no 1:58 done, no metabolic workup, no investigation into what was actually 2:00 investigation into what was actually 2:00 investigation into what was actually driving the decline. Carol didn't need 2:02 driving the decline. Carol didn't need 2:02 driving the decline. Carol didn't need someone to dismiss her symptoms. Carol 2:05 someone to dismiss her symptoms. Carol 2:05 someone to dismiss her symptoms. Carol needed someone to ask why. I'll come 2:08 needed someone to ask why. I'll come 2:08 needed someone to ask why. I'll come back to Carol in a minute, but first I 2:09 back to Carol in a minute, but first I 2:10 back to Carol in a minute, but first I need to explain what the research 2:11 need to explain what the research 2:11 need to explain what the research actually tells us about why this is 2:14 actually tells us about why this is 2:14 actually tells us about why this is happening and how far upstream it really 2:17 happening and how far upstream it really 2:17 happening and how far upstream it really begins. I want to challenge a belief 2:19 begins. I want to challenge a belief 2:19 begins. I want to challenge a belief that most people carrying into my 2:21 that most people carrying into my 2:21 that most people carrying into my office. The belief that cognitive 2:23 office. The belief that cognitive 2:23 office. The belief that cognitive decline is inevitable, that it just 2:26 decline is inevitable, that it just 2:26 decline is inevitable, that it just happens, that it runs in the family and 2:28 happens, that it runs in the family and 2:28 happens, that it runs in the family and there's nothing that can be done or that 2:30 there's nothing that can be done or that 2:30 there's nothing that can be done or that it's just aging. The published research 2:33 it's just aging. The published research 2:34 it's just aging. The published research tells a different story. Dr. Dr. Dale 2:37 tells a different story. Dr. Dr. Dale 2:37 tells a different story. Dr. Dr. Dale Bredesen, whose protocol I'm certified 2:39 Bredesen, whose protocol I'm certified 2:39 Bredesen, whose protocol I'm certified in, has identified more than 36 of these 2:43 in, has identified more than 36 of these 2:43 in, has identified more than 36 of these distinct metabolic factors that 2:45 distinct metabolic factors that 2:45 distinct metabolic factors that contribute to cognitive decline. But 36 2:48 contribute to cognitive decline. But 36 2:48 contribute to cognitive decline. But 36 addressable, measurable inputs that 2:52 addressable, measurable inputs that 2:52 addressable, measurable inputs that drive the process. That means cognitive 2:54 drive the process. That means cognitive 2:54 drive the process. That means cognitive decline is not a single disease. It's a 2:58 decline is not a single disease. It's a 2:58 decline is not a single disease. It's a cascade. The most common upstream
Sleep, Inflammation, and Metabolic Factors 3:00 cascade. The most common upstream 3:00 cascade. The most common upstream drivers that I see in clinical practice 3:03 drivers that I see in clinical practice 3:03 drivers that I see in clinical practice are chronic systemic inflammation 3:05 are chronic systemic inflammation 3:05 are chronic systemic inflammation markers like the high sensitivity CRP 3:08 markers like the high sensitivity CRP 3:08 markers like the high sensitivity CRP homocyine plus so many more. 3:11 homocyine plus so many more. 3:11 homocyine plus so many more. Inflammation damages that bloodb brain 3:14 Inflammation damages that bloodb brain 3:14 Inflammation damages that bloodb brain barrier that protective filter that 3:16 barrier that protective filter that 3:16 barrier that protective filter that keeps the toxins out of our brain. 3:18 keeps the toxins out of our brain. 3:18 keeps the toxins out of our brain. Another measure that I like is insulin 3:21 Another measure that I like is insulin 3:21 Another measure that I like is insulin resistance because that is huge against 3:23 resistance because that is huge against 3:23 resistance because that is huge against the brain. Some researchers call this 3:25 the brain. Some researchers call this 3:25 the brain. Some researchers call this Alzheimer's disease type three diabetes. 3:28 Alzheimer's disease type three diabetes. 3:28 Alzheimer's disease type three diabetes. I talked a lot about this in my week 3:30 I talked a lot about this in my week 3:30 I talked a lot about this in my week three that we just did last week. But 3:33 three that we just did last week. But 3:33 three that we just did last week. But these are some of the most important 3:35 these are some of the most important 3:35 these are some of the most important markers that we have. Another marker is 3:38 markers that we have. Another marker is 3:38 markers that we have. Another marker is hormones. Because our hormones decline, 3:41 hormones. Because our hormones decline, 3:41 hormones. Because our hormones decline, we get that atrophy. Estrogen, 3:43 we get that atrophy. Estrogen, 3:43 we get that atrophy. Estrogen, testosterone, and all those protective 3:46 testosterone, and all those protective 3:46 testosterone, and all those protective tissue hormones. So when that structure 3:48 tissue hormones. So when that structure 3:48 tissue hormones. So when that structure and that support and the integrity of 3:50 and that support and the integrity of 3:50 and that support and the integrity of the brain is going to be affected by 3:52 the brain is going to be affected by 3:52 the brain is going to be affected by this and cause decline other important 3:54 this and cause decline other important 3:54 this and cause decline other important things that need to be measured are 3:57 things that need to be measured are 3:57 things that need to be measured are nutrient deficiencies such as the 4:00 nutrient deficiencies such as the 4:00 nutrient deficiencies such as the homocyine level your B12 your folate 4:03 homocyine level your B12 your folate 4:03 homocyine level your B12 your folate your vitamin D because we know that 4:05 your vitamin D because we know that 4:05 your vitamin D because we know that things that happen to us chronically if 4:07 things that happen to us chronically if 4:07 things that happen to us chronically if we get chronic disease is either not 4:09 we get chronic disease is either not 4:09 we get chronic disease is either not having enough of what we're supposed to 4:11 having enough of what we're supposed to 4:11 having enough of what we're supposed to have or having too much of what that's 4:13 have or having too much of what that's 4:13 have or having too much of what that's affecting our brain. So we really need 4:15 affecting our brain. So we really need 4:15 affecting our brain. So we really need to look at both of those and measure 4:16 to look at both of those and measure 4:16 to look at both of those and measure those to see are you optimal. Another 4:19 those to see are you optimal. Another 4:19 those to see are you optimal. Another important factor for brain health and 4:21 important factor for brain health and 4:21 important factor for brain health and cognitive issues is your sleep 4:24 cognitive issues is your sleep 4:24 cognitive issues is your sleep architecture. Your brain clears out all 4:26 architecture. Your brain clears out all 4:26 architecture. Your brain clears out all this metabolic waste during that slow 4:28 this metabolic waste during that slow 4:28 this metabolic waste during that slow deep phase of sleep at night through a 4:31 deep phase of sleep at night through a 4:31 deep phase of sleep at night through a system called the glimpmphatic pathway. 4:33 system called the glimpmphatic pathway. 4:33 system called the glimpmphatic pathway. When sleep is fragmented that clearance 4:36 When sleep is fragmented that clearance 4:36 When sleep is fragmented that clearance doesn't happen and the proteins get ass 4:39 doesn't happen and the proteins get ass 4:39 doesn't happen and the proteins get ass that associated with cognitive decline 4:41 that associated with cognitive decline 4:41 that associated with cognitive decline accumulate. So all of these are not just 4:44 accumulate. So all of these are not just 4:44 accumulate. So all of these are not just peptide deficiencies. This is 4:46 peptide deficiencies. This is 4:46 peptide deficiencies. This is foundational work that we really need to 4:48 foundational work that we really need to 4:48 foundational work that we really need to look at. It's so important to address 4:51 look at. It's so important to address 4:51 look at. It's so important to address all these foundational things with a 4:53 all these foundational things with a 4:53 all these foundational things with a type of a functional medicine approach 4:55 type of a functional medicine approach 4:55 type of a functional medicine approach or just a good longevity approach of 4:59 or just a good longevity approach of 4:59 or just a good longevity approach of medicine. So it's just not replacing
Peptides for Neuroplasticity and Brain Repair 5:01 medicine. So it's just not replacing 5:01 medicine. So it's just not replacing peptides. And none of these are peptide 5:04 peptides. And none of these are peptide 5:04 peptides. And none of these are peptide deficiency specifically. The peptides 5:06 deficiency specifically. The peptides 5:06 deficiency specifically. The peptides come after they layer on top because 5:09 come after they layer on top because 5:09 come after they layer on top because once all these upstream environmental 5:11 once all these upstream environmental 5:11 once all these upstream environmental things are addressed, then that's the 5:13 things are addressed, then that's the 5:14 things are addressed, then that's the time to start layering in the peptides. 5:16 time to start layering in the peptides. 5:16 time to start layering in the peptides. And that is the difference between 5:18 And that is the difference between 5:18 And that is the difference between symptom management and longevity 5:19 symptom management and longevity 5:19 symptom management and longevity medicine. So, I promised you in about 5:22 medicine. So, I promised you in about 5:22 medicine. So, I promised you in about Carol's story, and I'm going to get 5:24 Carol's story, and I'm going to get 5:24 Carol's story, and I'm going to get there, but in the next few minutes, I'm 5:25 there, but in the next few minutes, I'm 5:26 there, but in the next few minutes, I'm going to show you exactly what cognitive 5:28 going to show you exactly what cognitive 5:28 going to show you exactly what cognitive peptides do and why the sequence 5:31 peptides do and why the sequence 5:31 peptides do and why the sequence matters. So, stay with me. Once we've 5:34 matters. So, stay with me. Once we've 5:34 matters. So, stay with me. Once we've addressed again all these upstream 5:36 addressed again all these upstream 5:36 addressed again all these upstream drivers, there is a category of peptides 5:38 drivers, there is a category of peptides 5:38 drivers, there is a category of peptides that directly support the brain's 5:40 that directly support the brain's 5:40 that directly support the brain's capacity to repair, protect, and that 5:43 capacity to repair, protect, and that 5:44 capacity to repair, protect, and that neuroplasticity. 5:45 neuroplasticity. 5:45 neuroplasticity. Neurosplasticity is your brain's ability 5:48 Neurosplasticity is your brain's ability 5:48 Neurosplasticity is your brain's ability to form the new connections, adapt, and 5:52 to form the new connections, adapt, and 5:52 to form the new connections, adapt, and recover. It doesn't stop at age 30 or 50 5:56 recover. It doesn't stop at age 30 or 50 5:56 recover. It doesn't stop at age 30 or 50 or 70, but it does require the right 5:58 or 70, but it does require the right 5:58 or 70, but it does require the right environment to do its job. The first is 6:01 environment to do its job. The first is 6:01 environment to do its job. The first is CAX. Cax is a synthetic analog of a 6:04 CAX. Cax is a synthetic analog of a 6:04 CAX. Cax is a synthetic analog of a natural brain peptide derived from act. 6:09 natural brain peptide derived from act. 6:09 natural brain peptide derived from act. That's a hormone that your pituitary 6:11 That's a hormone that your pituitary 6:11 That's a hormone that your pituitary produces and signals to the body and the 6:13 produces and signals to the body and the 6:13 produces and signals to the body and the brain. The primary mechanism is 6:16 brain. The primary mechanism is 6:16 brain. The primary mechanism is increasing the BDNF. BDNF stands for 6:19 increasing the BDNF. BDNF stands for 6:19 increasing the BDNF. BDNF stands for brain derived neurotrophic factor. Think 6:23 brain derived neurotrophic factor. Think 6:23 brain derived neurotrophic factor. Think of B&D BDNF as the fertilizer for the 6:26 of B&D BDNF as the fertilizer for the 6:26 of B&D BDNF as the fertilizer for the neurons. It enhances focus and the 6:29 neurons. It enhances focus and the 6:29 neurons. It enhances focus and the processing speed and has been studied 6:31 processing speed and has been studied 6:31 processing speed and has been studied for neuroproction after brain injury. In 6:34 for neuroproction after brain injury. In 6:34 for neuroproction after brain injury. In clinical use, CAX is typically used 6:37 clinical use, CAX is typically used 6:38 clinical use, CAX is typically used intraasally, meaning it's is delivered 6:40 intraasally, meaning it's is delivered 6:40 intraasally, meaning it's is delivered through the nose mucosa which goes 6:42 through the nose mucosa which goes 6:42 through the nose mucosa which goes directly to the brain through the 6:43 directly to the brain through the 6:44 directly to the brain through the cribopform plate. The second one I like 6:46 cribopform plate. The second one I like 6:46 cribopform plate. The second one I like to talk about is salac. Salank is an 6:49 to talk about is salac. Salank is an 6:49 to talk about is salac. Salank is an angiolytic peptide meaning it reduces 6:52 angiolytic peptide meaning it reduces 6:52 angiolytic peptide meaning it reduces anxiety without sedation. It modulates 6:56 anxiety without sedation. It modulates 6:56 anxiety without sedation. It modulates the GABA hormone. That's the brain's 6:58 the GABA hormone. That's the brain's 6:58 the GABA hormone. That's the brain's calming neurotransmitter and the 7:01 calming neurotransmitter and the 7:01 calming neurotransmitter and the serotonin pathways. Clinical research 7:04 serotonin pathways. Clinical research 7:04 serotonin pathways. Clinical research has shown improvements in memory, 7:06 has shown improvements in memory, 7:06 has shown improvements in memory, attention, mental clarity alongside 7:09 attention, mental clarity alongside 7:09 attention, mental clarity alongside reducing that anxiety. The third is 7:12 reducing that anxiety. The third is 7:12 reducing that anxiety. The third is Dhexa. Dhexa is one of the most powerful 7:16 Dhexa. Dhexa is one of the most powerful 7:16 Dhexa. Dhexa is one of the most powerful potent neuroplasticity supporting 7:18 potent neuroplasticity supporting 7:18 potent neuroplasticity supporting compounds. It works by increasing what 7:20 compounds. It works by increasing what 7:20 compounds. It works by increasing what we call the HGF/CMT 7:23 we call the HGF/CMT 7:23 we call the HGF/CMT signaling pathway which drives
Gut-Brain Axis and Cognitive Health 7:25 signaling pathway which drives 7:25 signaling pathway which drives synaptogenesis, the formation of new 7:29 synaptogenesis, the formation of new 7:29 synaptogenesis, the formation of new connections between neurons. This early 7:32 connections between neurons. This early 7:32 connections between neurons. This early research suggests it may be millions of 7:34 research suggests it may be millions of 7:34 research suggests it may be millions of times more potent than even BDNF at 7:37 times more potent than even BDNF at 7:37 times more potent than even BDNF at promoting synaptic growth. One more 7:39 promoting synaptic growth. One more 7:39 promoting synaptic growth. One more peptide worth naming here, and if you 7:41 peptide worth naming here, and if you 7:41 peptide worth naming here, and if you watched week two, you've already met it. 7:44 watched week two, you've already met it. 7:44 watched week two, you've already met it. The BPC57 which I covered in the context 7:47 The BPC57 which I covered in the context 7:47 The BPC57 which I covered in the context of healing and gut gut repair. It also 7:51 of healing and gut gut repair. It also 7:51 of healing and gut gut repair. It also plays a role in the gut brain axis. Your 7:53 plays a role in the gut brain axis. Your 7:54 plays a role in the gut brain axis. Your gut produces approximately 90% of your 7:56 gut produces approximately 90% of your 7:56 gut produces approximately 90% of your body's serotonin. Your vagus nerve 7:59 body's serotonin. Your vagus nerve 7:59 body's serotonin. Your vagus nerve carries a signal directly between your 8:01 carries a signal directly between your 8:01 carries a signal directly between your gut and your brain. So when the gut is 8:04 gut and your brain. So when the gut is 8:04 gut and your brain. So when the gut is inflamed or the mucosal lining is 8:06 inflamed or the mucosal lining is 8:06 inflamed or the mucosal lining is compromised of your gut, that 8:08 compromised of your gut, that 8:08 compromised of your gut, that communication becomes disrupted. BPC57's 8:13 communication becomes disrupted. BPC57's 8:13 communication becomes disrupted. BPC57's healing's effects on the gut lining has 8:15 healing's effects on the gut lining has 8:15 healing's effects on the gut lining has these downstream implications for 8:17 these downstream implications for 8:17 these downstream implications for neurotransmitter production and the 8:20 neurotransmitter production and the 8:20 neurotransmitter production and the inflammatory load reaching your brain. 8:23 inflammatory load reaching your brain. 8:23 inflammatory load reaching your brain. So cognitive health is not just a brain 8:26 So cognitive health is not just a brain 8:26 So cognitive health is not just a brain problem, it's a systems problem and the 8:29 problem, it's a systems problem and the 8:29 problem, it's a systems problem and the treatment has to match that. Let's get
Carol's Test Results and Treatment Plan 8:31 treatment has to match that. Let's get 8:31 treatment has to match that. Let's get back to Carol. When I ran her 8:33 back to Carol. When I ran her 8:33 back to Carol. When I ran her comprehensive panel, here's what we 8:35 comprehensive panel, here's what we 8:35 comprehensive panel, here's what we found. Her homocyine was significantly 8:38 found. Her homocyine was significantly 8:38 found. Her homocyine was significantly elevated. This is one of the mo 8:40 elevated. This is one of the mo 8:40 elevated. This is one of the mo strongest modifiable risk factors for 8:42 strongest modifiable risk factors for 8:42 strongest modifiable risk factors for cognitive decline. 8:44 cognitive decline. 8:44 cognitive decline. Her high sensitivity CRP was elevated. 8:47 Her high sensitivity CRP was elevated. 8:47 Her high sensitivity CRP was elevated. Her vitamin D was deficient, which we 8:49 Her vitamin D was deficient, which we 8:50 Her vitamin D was deficient, which we know is so important for energy and 8:51 know is so important for energy and 8:51 know is so important for energy and immune. Her B12 was at a very bottom of 8:54 immune. Her B12 was at a very bottom of 8:54 immune. Her B12 was at a very bottom of the reference range. And her fasting 8:57 the reference range. And her fasting 8:57 the reference range. And her fasting insulin was elevated, meaning early 8:59 insulin was elevated, meaning early 8:59 insulin was elevated, meaning early insulin resistance. A home sleep study 9:02 insulin resistance. A home sleep study 9:02 insulin resistance. A home sleep study showed severely fragmented sleep. She 9:05 showed severely fragmented sleep. She 9:05 showed severely fragmented sleep. She was never reaching that slow deep wave 9:07 was never reaching that slow deep wave 9:07 was never reaching that slow deep wave sleep that cleans out the brain. Carol 9:10 sleep that cleans out the brain. Carol 9:10 sleep that cleans out the brain. Carol did not have stress. Carol did not have 9:13 did not have stress. Carol did not have 9:13 did not have stress. Carol did not have permenopause related brain fog. Carol 9:16 permenopause related brain fog. Carol 9:16 permenopause related brain fog. Carol had six measurable treatable upstream 9:20 had six measurable treatable upstream 9:20 had six measurable treatable upstream drivers of cognitive decline. None of 9:22 drivers of cognitive decline. None of 9:22 drivers of cognitive decline. None of which had ever been looked at before. We 9:25 which had ever been looked at before. We 9:25 which had ever been looked at before. We built her protocol. We corrected the 9:27 built her protocol. We corrected the 9:27 built her protocol. We corrected the homoyine with targeted B vitamins. We 9:30 homoyine with targeted B vitamins. We 9:30 homoyine with targeted B vitamins. We targeted and addressed her vitamin D 9:33 targeted and addressed her vitamin D 9:33 targeted and addressed her vitamin D deficiency, her diet, her sleep 9:35 deficiency, her diet, her sleep 9:35 deficiency, her diet, her sleep architect. Then is when we added the CJC 9:39 architect. Then is when we added the CJC 9:39 architect. Then is when we added the CJC 1295 and epipomor to support her growth 9:42 1295 and epipomor to support her growth 9:42 1295 and epipomor to support her growth hormone pulse and the slowwave sleep. We 9:45 hormone pulse and the slowwave sleep. We 9:45 hormone pulse and the slowwave sleep. We addressed her insulin resistance through 9:47 addressed her insulin resistance through 9:47 addressed her insulin resistance through diet and lifestyle factors. And once 9:49 diet and lifestyle factors. And once 9:49 diet and lifestyle factors. And once that foundation was in place, we added 9:52 that foundation was in place, we added 9:52 that foundation was in place, we added the CAX. At 60 days, Carol told me that 9:55 the CAX. At 60 days, Carol told me that 9:55 the CAX. At 60 days, Carol told me that the fog began to lift. At 90 days, she 9:59 the fog began to lift. At 90 days, she 9:59 the fog began to lift. At 90 days, she was finishing her sentences again. And
Recovery and Final Takeaways 10:01 was finishing her sentences again. And 10:01 was finishing her sentences again. And at 6 months, she came back in and said, 10:04 at 6 months, she came back in and said, 10:04 at 6 months, she came back in and said, "I feel like I got my brain back." This 10:07 "I feel like I got my brain back." This 10:07 "I feel like I got my brain back." This is the result of testing before 10:09 is the result of testing before 10:09 is the result of testing before treating. Or even asking why, not just 10:13 treating. Or even asking why, not just 10:13 treating. Or even asking why, not just what. And it's about understanding that 10:15 what. And it's about understanding that 10:16 what. And it's about understanding that the brain is the most complex organ in 10:18 the brain is the most complex organ in 10:18 the brain is the most complex organ in the body and it deserves a protocol that 10:21 the body and it deserves a protocol that 10:21 the body and it deserves a protocol that matches that complexity. I want to pause 10:23 matches that complexity. I want to pause 10:23 matches that complexity. I want to pause here because Carol's story is one that I 10:26 here because Carol's story is one that I 10:26 here because Carol's story is one that I hear almost every week in my practice. 10:29 hear almost every week in my practice. 10:29 hear almost every week in my practice. Someone who knew something was wrong, 10:31 Someone who knew something was wrong, 10:31 Someone who knew something was wrong, who said, and she even was saying it out 10:33 who said, and she even was saying it out 10:33 who said, and she even was saying it out loud to her physicians. She was just 10:36 loud to her physicians. She was just 10:36 loud to her physicians. She was just sent home with the word normal with no 10:39 sent home with the word normal with no 10:39 sent home with the word normal with no real answers. So, I want to ask you 10:42 real answers. So, I want to ask you 10:42 real answers. So, I want to ask you something. Have you ever been dismissed 10:44 something. Have you ever been dismissed 10:44 something. Have you ever been dismissed for a symptom that you knew was real? 10:47 for a symptom that you knew was real? 10:47 for a symptom that you knew was real? Maybe it's brain fog. It could be 10:50 Maybe it's brain fog. It could be 10:50 Maybe it's brain fog. It could be difficulty finding that word, energy 10:53 difficulty finding that word, energy 10:53 difficulty finding that word, energy collapse, poor sleep, just overall 10:55 collapse, poor sleep, just overall 10:55 collapse, poor sleep, just overall inflammation, feeling. If so, did did 10:59 inflammation, feeling. If so, did did 10:59 inflammation, feeling. If so, did did you just keep pushing for answers or did 11:00 you just keep pushing for answers or did 11:00 you just keep pushing for answers or did you just start believing that what they 11:02 you just start believing that what they 11:02 you just start believing that what they told you was normal? Drop your answer in 11:05 told you was normal? Drop your answer in 11:05 told you was normal? Drop your answer in the comments because I read every one of 11:06 the comments because I read every one of 11:06 the comments because I read every one of them. And if you're still looking for 11:08 them. And if you're still looking for 11:08 them. And if you're still looking for answers, let's talk in the comments. I'd 11:11 answers, let's talk in the comments. I'd 11:11 answers, let's talk in the comments. I'd love to discuss this more online. I also 11:13 love to discuss this more online. I also 11:13 love to discuss this more online. I also want you to check out all my resources 11:15 want you to check out all my resources 11:15 want you to check out all my resources that we link down below in the 11:16 that we link down below in the 11:16 that we link down below in the description because those there's so 11:18 description because those there's so 11:18 description because those there's so much information that I'd love to share 11:20 much information that I'd love to share 11:20 much information that I'd love to share with you. It's all free. So, I want to 11:23 with you. It's all free. So, I want to 11:23 with you. It's all free. So, I want to thank you for being here because 11:24 thank you for being here because 11:24 thank you for being here because everything I do, every protocol, every 11:27 everything I do, every protocol, every 11:27 everything I do, every protocol, every video, every conversation runs through 11:29 video, every conversation runs through 11:29 video, every conversation runs through the same belief. I want to help you live 11:32 the same belief. I want to help you live 11:32 the same belief. I want to help you live smarter, stronger, longer because I 11:36 smarter, stronger, longer because I 11:36 smarter, stronger, longer because I don't want your first symptom being your 11:38 don't want your first symptom being your 11:38 don't want your first symptom being your last. We'll see you next week.
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