Overcome Your Infertility Challenges

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Summary
  • Understanding the individual’s hormonal profile and gene expression is crucial in addressing infertility challenges
  • Androgen dominance, characterized by high testosterone levels and inefficient conversion of testosterone into estrogen, can contribute to infertility issues
  • Personalized supplementation and hormone management based on gene expression can help optimize hormone levels and improve fertility outcomes
Transcript
Kashif Khan

Hello everyone, and welcome back for another case study. We’re talking about an actual patient and how they were able to resolve their infertility issues, which is a really cool story. Now, keep in mind that if you didn’t watch the previous case studies, in order to protect patient confidentiality, we’re not using their report. We’re using mine. We don’t want their name to show up. It shows up on the report; we’re using my report. We found somebody who has the same genetic profile as being in a particular area, and we asked their permission to tell their story so I could use my report to show you what that profile looks like. We’re going to look into hormones to show you how we were able to resolve somebody’s root-cause infertility issues. If I go through the portal now and dig into the hormone section, when I go to the summary, I’m going to show you, first of all, the profile they had, and then we’ll speak to what was going on. Like me, they were androgen-dominant. This is the avatar. It’s not a gene, but if you take a collection of hormone genes, the cascade, and add them together, you get me and also this patient, who have androgen dominance, meaning they lean more on the testosterone side. Why is that happening? The fast conversion of progesterone into testosterone produces a heavy pool of testosterone. The hormone levels are high to begin with, with an increase in binding, the ability to utilize the androgen receptor, and the ability to actually utilize that testosterone very efficiently. Then you see the slow conversion of androgen into estrogen.

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