Full Transcript 0:00 Well, A.J. 0:00 Lanigan, it's always such a pleasure to get to chat with you. 0:04 I feel smarter anytime I sit and in the vicinity of you. 0:08 So thank you so much for being here with me today. 0:10 Well, I appreciate you. 0:11 I feel like I should hang around with smart people like you all the time. 0:15 I love it, I love it, and one thing that you know, 0:18 when you deal with cancer, you think of immune system. 0:20 I mean, you you think, you know, 0:23 at the end of the day, you look upon that as a police force 0:26 that is going to be out there checking, making sure that no one wanted 0:30 cancer cells, you know, settles in, create communities and start to build cities. 0:35 And so what can you tell me? 0:38 I mean, you have the the adaptive immune system. 0:41 You have the innate immune system. 0:43 It can you just give a little bit of an overview. 0:46 What immune system actually contains of. 0:49 So when we use that phrase, people are a little bit 0:51 more of an understanding of what they're dealing with. 0:54 Yeah, I think I think a little bit of a primer, 0:56 even for those that maybe advanced and immunology, bear with us a moment, 1:02 but but I like to start off by saying, you know, we've got two arms 1:06 to the immune system, the innate that which we had 1:09 when we drew our first breaths the day we were born. 1:13 And then we have the acquired or I sometimes refer to it 1:17 as the learned or learned side of the immune system, 1:20 where once we come into contact with something, 1:25 then it learns it, remembers it 1:28 with a specific memory. 1:31 Whereas on the innate side, typically we we've thought of it 1:36 for so many years as it's either self or it's non-self. 1:42 And if it's non-self, we just can't open up 1:46 a can of, we'll pass on it and get rid of it and 1:50 go back to at rest. 1:52 That's kind of a broad a broad case. 1:55 Now at some point we may want to touch on something that's happened 1:58 over the last ten years or so, which is called trim or trained immunity. 2:03 But to start off with, let's just establish that we've got those two arms. 2:08 Now, there's also two other things 2:11 we need to think of in regards to the being system. 2:14 We have those immune cells and their activity 2:18 which are actually involved in the killing. 2:21 Okay. 2:22 We have this other part of the immune system 2:25 that's more into reconnaissance tagging things. 2:30 This is where antibodies come in. 2:32 Antibodies are certainly important. 2:35 They don't do the best job of killing, but they do a great job of tagging things, 2:41 helping the other side of the immune system locate them 2:46 quickly, and eliminating hopefully before they cause us any challenges. 2:51 I know that a tremendous amount of your patient population is vexed with cancer, 2:57 and that's certainly a big part of what the immune system, disease itself. 3:02 I always like to tell people the best way 3:05 to deal with cancer, of course, is not to get it. 3:09 So I'm a big proponent of getting way ahead of the curve, 3:13 even down with the young children, getting those immune systems 3:18 properly supported throughout their life 3:22 to raise their odds. 3:24 You know the statistic, depending on who you talk to, 3:27 one out of three men, one out of two, one out of two men, one out of three, one. 3:31 Whoever's keeping track will be challenged 3:35 by cancer sometime in their life. 3:38 I'd like to cheat a little bit on that. 3:41 I like to get some odds more in my favor. 3:44 So that's kind of where we want to steer today. 3:46 Is is recognizing this immune system is incredible. 3:51 And as marvelous as it is, we should do what we can to support it 3:56 and enhance its already miraculous abilities and not do the things 4:02 that are going to suppress it, depress it, and knock it out. 4:08 So. So essentially what I'm hearing from you is that you're 4:11 you're dealing with two aspects. 4:12 You have the the information gathering and information transport. 4:17 So to say that that's one aspect. 4:19 And then you have the aspect where you're responding 4:22 to that information and then going out to to kill. 4:26 So and obviously you are going to be in a much better place 4:31 if you're able to respond to things faster 4:34 and have better intelligence available sooner. 4:38 Kind of like you have two countries and you have a border control, 4:41 and these two countries are at odds with each other. 4:44 And if we allow them things, you know, people from the one country 4:49 just come in and start to set up big cities, you know, 4:53 then it's going to be harder to get them out of that country versus 4:58 if we have an immediate response to at the border, say, hey, 5:02 you don't belong here, you don't have a passport, you need to get out of here. 5:06 Or if they go and disguise and to have, you know, a secret agents 5:10 that are able to recognize that faster and then bring that to the local police 5:14 or local agencies. 5:16 So to say, 5:16 to kind of clear them out faster, is that kind of what I'm hearing from you? 5:20 Yeah. I've got a tech pointed 5:24 or these different things 5:27 that exist on both the immune cells and the cancer. 5:31 If the cancer cells hijack as an example, 5:36 these checkpoints, they'll keep the the traffic light red. 5:41 They'll stop our immune system altogether 5:44 even though the hordes are coming. 5:47 And so now there are different forms of immunotherapy 5:52 that have all developed, by the way, through our 5:55 our life to. 5:56 But if we can't, if we get an understanding that there's nothing fair 6:01 about the way that cancer deals with us, 6:05 then we don't need to be fair with it. 6:09 That's why, as we bring into the conversation a little bit, 6:13 our beta glucan ingredients starting 6:16 not not after the cows over the hill, 6:20 but before 6:22 the adult has an opportunity to have a cow. 6:26 Okay, we we want to start with them when they're in kindergarten, like, 6:31 you know, three, four years old, eating the right food, drinking the good 6:36 water, breathing the good air, getting proper rest, getting proper exercise. 6:41 All of these things are critically important. 6:43 But what we can do that the good diet and these other things cannot 6:49 and will not do is have a specific effect 6:52 on our immune cells, causing them to do what 6:56 they're supposed to do, but more, better and faster. 6:59 We could get into the tall weeds on that, but that's from this 50 7:03 600 zero square feet that that's what we're all about. 7:07 So and 7:08 because, I mean, it sounds like the I mean, because that immediate response 7:12 or more immediate response is our innate immune system. 7:16 Yeah. 7:16 And and like you're saying, I mean, now researchers come out 7:22 showing that innate immune system, which is that kind of the media response 7:26 which we didn't realize could be trained. 7:29 Now we understand that it can be trained. 7:32 Can you talk a little bit about that research? 7:36 20 or 30 years ago, 7:39 the immune cell that's actually 7:41 the most abundant, the neutrophils. 7:44 When you do blood work you get a panel and the any you. 7:48 So those are your neutrophils. 7:50 And they give you basically a gross count. 7:53 How many do you have. 7:54 And there's a range that we consider to be normal. 7:59 That's based on these bell shaped curves where if you're dead in the middle 8:02 of all the people that we looked at, that's perfect. 8:06 But some people may just naturally have a higher level. 8:10 Some people may have a naturally lower level. 8:14 In regards to count. 8:17 The big 8:17 question that I asked, and this is where we start to get in the tall 8:21 weeds, is how well are those neutrophils functioning? 8:25 I used the example of going into 8:27 the Empire State Building and counting how many light bulbs there are. 8:31 Well, that's an interesting number. 8:33 There's going to be thousands, possibly tens of thousands. 8:36 But how many of those light bulbs are working properly? 8:40 So the next time we do blood work we look at the cell counts. 8:45 But we also need to ask how well are those numbers functioning. 8:50 And so neutrophil function looking at phagocyte 8:56 or phagocytosis or the phagocytic index. 9:00 As an example we look at other numbers. 9:03 We look at monocytes MONO. 9:06 We look at natural killer cells. 9:09 Very important. 9:10 But there is a natural killer cells function test. 9:14 We can go down the list. 9:16 But the general numbers tell us. 9:20 Oh you've got a lot of immune cells going on there. 9:23 But it's that next layer. 9:26 And the layer after that when we start getting down 9:29 to, hey, I've got a great army assembled here, 9:33 but how many of them are laying in the bunks asleep? 9:36 How about how many of them are otherwise impaired? 9:39 And your immune system must recognize and respond to that picture, 9:45 however it's being offered to 9:48 then provide us with the proper antibodies. 9:52 Now again, we're dealing with the immune system. 9:54 Now that works in a specific form. 9:58 One shot, one target, 10:02 a second shot, a second target. 10:07 Not like the innate side. 10:09 The innate said says, I'm going to surveil, I'm going to identify. 10:14 I'm going to if it doesn't identify properly, 10:18 I'm going to kill, and I'm going to eliminate. 10:21 Those are the four things that our immune system generally has to do. 10:28 One just does it differently. 10:29 One does it rapidly and helps you to survive. 10:33 That's another thing. 10:36 The acquired side of the immune system 10:39 takes days and door weeks 10:42 to mount this specific response. 10:46 The innate side we're talking about minutes, hours in a day or so. 10:51 You get a splinter in your big toe while you're out there grounding. 10:56 Okay, next morning, if you don't get that splinter out, 10:59 what you you've got redness, you've got pus. 11:03 That's what you don't want. 11:05 A cancer cell shows up if a cell starts getting screwy, 11:10 if a group of cells start going sideways and not acting the way 11:15 that they're supposed to, they may not be cancerous. 11:18 It may not even be precancerous yet, but you don't want them in a 11:23 and immune cell that's functioning. 11:26 Or a group of immune cells that are are responding properly, competently, 11:33 are going to 11:33 knock out that cell, get rid of it, that cell will be properly replaced. 11:37 And you live a long life and you look good doing it. 11:42 And this is 11:43 the Nate that has that immediate response. 11:47 And but the the thought in the past was 11:50 that it's kind of like it learned 11:53 as it was going and and then it forgot for the next attack 11:57 and then whatever memory was passed on to the adaptive. 12:00 But the innate didn't have any memory. 12:03 But but we're understanding now that even the innate, 12:07 you know, the faster responder now can also have a trained aspect 12:12 so that they faster can recognize what they for future 12:17 attacks, future fast attacks that they can respond to that. 12:21 Imagine imagine a group of highly 12:25 qualified soldiers 12:28 have a book in front of them 12:30 that when non-self shows up, 12:33 they typically go through the book, do step one, step two. 12:37 And there are multiple steps, by the way, depending on if you're neutrophil 12:41 or you're a natural killer cells 12:43 or you're a monocyte or you're a macrophage, there's a book 12:47 and the book gets open to certain pages and certain things get 12:50 done in proper order. 12:53 We takes the same book 12:56 and it puts some post-it notes in 1 or 2 places 13:01 so that when something comes 13:04 a second time or a third time, they don't have to go through page one 13:09 through 15, they can get right to the page 13:13 where they start nailing this target. 13:17 And so we're actually talking about here epigenetic changing. 13:21 We're shortening the book and the cliff notes. 13:25 Not everybody used cliff notes I would use them occasionally. 13:29 Can I say I read every single page of the Iliad and Odyssey? 13:34 No, but I, I certainly wore out a pair of cliff notes. 13:39 Same thing with Moby Dick. 13:41 I still was able to do well 13:45 when it came time to write the book report. 13:48 So what we're doing is we're getting 13:51 the odds more 13:52 on our side when we take that innate immune system 13:57 and we get it to do the same thing 14:01 that it was designed to do. 14:02 Our material is not going to make it do something. 14:04 It's not supposed to be doing that. 14:07 That might lead to auto immune disorders and so forth. 14:11 We don't want that. 14:13 These changes, doc, go all the way 14:16 back up into including the bone marrow 14:20 after a certain number of weeks, actually months. 14:23 Because again, this process of trained immunity 14:26 happens over a period of time after around 3 to 4 months. 14:30 And we've never really nailed down a particular number of days or weeks. 14:34 But we know it takes time to establish trained immunity. 14:40 We've got modest sites and so forth, coming right out of the bone 14:43 marrow that have this trained immunity. 14:46 Meaning overall, 14:50 I mean, response is better. 14:52 This innate side cooperates 14:56 fully with the acquired side of the immune system. 15:00 You you have this group of cells called dendritic cells. 15:05 And as the macrophages and neutrophils, they're kind of sloppy 15:09 in the way that they eat. 15:10 And they leave little tidbits little crumbs 15:13 of the pathogen the added genetic marker. 15:18 And they grab it with these tentacles. 15:21 And they run over through the lymph nodes. 15:24 And they present it to various T cells. 15:28 And then the T cells says oh well I don't recognize that. 15:32 They'll present it to another and another. 15:34 And finally we'll have in the population 15:38 of one or more that will embrace it and say, that's important, 15:42 I need to teach this now to my B cells, 15:46 to create plasma cells, to make antibodies, because 15:51 both sides of the 15:52 immune system downstream are going to now work together 15:56 and give us a more global chance, 15:59 a better chance, better odds to overcome, whether it's cancer, 16:04 whether it's influenza, whether it's whatever Covid is, you know, 16:09 these are all things that living here on this planet 16:13 are going to continue to challenge us. 16:15 And if we're living here on this planet, we need all the odds 16:19 we can get moved in our favor. 16:22 So even the bone marrow. 16:24 So when you're talking 16:25 about the trained immunity, you know, the innate trained immunity, 16:29 then even the cells that are then produced in the bone marrow. 16:33 So they're actually epigenetic changes, like you're saying they're, 16:36 you know, you have these genes are switched on, switched off. 16:39 And from my understanding, there's also kind of how the 16:43 the immune cells, how they gain energy is shifting. 16:47 And that also triggers some of these epigenetic changes within that cell. 16:51 And now because of those switches within your gene, 16:55 how they're turned on, turned off are now optimized 16:58 within those immune cells to go after these offenders. 17:03 You know, if they are there again, you know that those immediate attack 17:07 immune cells and that actually so these cells that are produced 17:11 and in the bone marrow, the white blood cells, they are actually now ready to go. 17:15 They have the perfect training, perfect switches turned on, turned off 17:19 so that when a cancer cell that has been, like you said, it's been identified. 17:25 Now they can attack that, you know, the, you know, much faster. 17:29 It speed is speed is is the key. 17:33 Just like with an American football, if you've got the fastest runner, 17:37 they're going to dart right past the defense. 17:40 If you've got a fast linebacker, a fast defensive back, 17:44 the defensive back is going to intercept the pass. 17:46 That blitzing linebacker is going to sack the quarterback. 17:49 We want speed, 17:53 which is part of the effectiveness part. 17:55 If it normally would take, let's say three days for a series of events 17:59 to occur, we won't have to happen in one day again. 18:04 When I, I keep coming back, we're going to win the battle against cancer. 18:09 Not by playing fair, not by, you know, a flat playground. 18:14 We want to pile on as much as we can pile on 18:18 to make sure that the cancer cells don't have a chance. 18:23 You spend so much of your time with people that have run the gantlet. 18:28 Maybe they've been through chemo, radiation, surgery, 18:32 a conglomeration of 2 or 3. 18:34 Who knows? 18:36 Gosh, would it be great if met those people years 18:39 before and say, look, 18:42 I'm going to read the future for you and lay out the statistics 18:46 in our message back over the hill before the devil pops up. 18:53 Yeah, that's the thing. 18:54 Like you're saying, cancer doesn't fight fair and so why should we? 18:58 And then you talk about glue. 19:00 Can you know in in regards to our ability then to upgrade our game 19:06 and be able to speed up that recognition, speed up 19:09 that training, all that innate immune system. 19:13 So, so talk to me a little bit how beta glue can play a role 19:18 in speeding up the, you know, the training and the memory of the innate. 19:23 Our sales go up. 19:25 I don't reject that. 19:26 But that's not what we do now. 19:29 It is a complex carbohydrate, the material. 19:32 And if you take enough of it, it definitely will affect 19:35 your cholesterol numbers and so forth. 19:38 But our main ingredient comes from the cell wall of what 19:44 you would know as a Bakers East, specifically Saccharomyces cerevisiae. 19:49 And beyond that, a specific strain of that material. 19:53 Now the Chinese, the Japanese, the other Asian populations 19:59 have been telling 20:00 stories about mushrooms for give or take, 3000 years. 20:04 And yes, all fungal forms and mushrooms are fungal forms. 20:09 So is smut moles, yeast, which we we we bring it from yeast. 20:16 All of these have glucans, 20:19 but every form, every source 20:23 has a different molecular structure. 20:26 They have a different molecular weight. 20:29 They have different side branching. 20:32 Some from say you Gleaner 20:35 only has a one three backbone, where the others may be 20:40 a variable 1314 glucan chain. 20:45 That's going to be your cereal grains. 20:48 Our material has at one three 20:52 backbone. 20:54 And every so many 20:57 glucose molecules there'll be a side chain, that one six side chain 21:02 with another little short one three glucan tail dropping. 21:07 Now a lot of people will say, but I want to say for sure, 21:11 I did choose this stuff, okay, 21:14 the immune system chose this molecule 21:18 or this fake source, Saccharomyces cerevisiae. 21:23 And of all the different strains out there, this particular strain, 21:27 and it says of all of it out there, 21:30 we will bind to this one. 21:32 We will recognize this, this one better than all the others they think of. 21:38 And we're talking now about a receptor called deck and one deck. 21:43 That one is a very important protein that's on the source of sales 21:47 and not just one. 21:48 There's a bunch of kind of like a hairy porcupine. 21:51 And when one of them really gets a good grip, 21:55 then it will 21:55 start to attract other deck and ones to that same area 22:00 and form a what we call a phagocytic area that, that, that starts to engulf 22:07 and pull that beta glucan particle in. 22:11 Now use the term particle because there are water soluble versions 22:17 of the same material 22:20 with all these different sources. 22:22 And it's not that it doesn't recognize it. 22:26 It just gives kind of a weak handshake, the kind of handshake that you 22:30 you really can't embrace. This is what you want. 22:33 And that's what you get with this particular material. 22:36 Now there's a second receptor 22:39 that for the first 20 years I've been at this now 30 years 22:42 for the first 20 years, we talked about complement 22:46 receptor number three. 22:49 Now think of it as a dual system. 22:53 The one side will embrace the water soluble molecule. 23:00 But then 23:01 there's got to be a target that has been opsin or something 23:05 stuck to it called complement 23:08 specifically C three. 23:11 There's an enzyme C three convertase that splits it in half. 23:16 C3A goes down the road and leaves 23:20 red markers to bring other immune cells. 23:23 The C three, B six. 23:25 They're kind of like chewing gum to the soul of your foot. 23:28 And in the literature you'll see I see 23:31 3BI means inactivated. 23:34 So you've got a target that's been verified, 23:38 a cell that's been invaded by a virus, viruses. 23:44 They don't do well out in the open. 23:46 Turn it into Looney Tunes so that everybody can maybe understand 23:50 it a little better and embrace it. 23:53 So. So you have the ones essentially you have the fungal 23:57 beta glucans that you're talking about from the east, 24:01 and then they can bind them to detect and one 24:04 and then they that initiates and that receptor signaling. 24:08 And from my understanding that shifts 24:12 a kind of the changes how that cell produces energy 24:17 and that changes and different cellular signaling and epigenetic changes 24:23 within that cell, that then modifies how that cell is going to respond 24:29 in the future to be faster and how it's responding. 24:35 The epigenetic 24:36 change doesn't happen on first contact. 24:40 Epigenetic change happens over time. 24:42 This is this is one of the things that I've continued to try to explain to 24:46 people. 24:47 It's not like taking two aspirin and your headache is gone in 30 minutes. 24:51 No, it takes time. 24:53 Now what will happen? 24:56 The immune response, in other words, the degree. 24:59 And again let's go back to neutrophils because they are critically important. 25:04 Whoever made our immune systems gave us more neutrophils than anything else. 25:09 So let's talk about those we can actually measure. 25:13 And this has been done hundreds and hundreds of times. 25:16 Published in the literature. 25:18 We give salt water phosphate buffered saline as a base. 25:23 And let's say 30% of our neutrophil will respond to a challenge. 25:29 Okay. 25:30 We can increase that from 30% 25:34 to 35%, 40%, 45%, and so forth. 25:41 By increasing the dose, every time we increase the dose, 25:45 we get a a higher immune response. 25:49 Now, even from first dose, it takes whatever 25:52 that dose is, whether you're taking one X or whatever you're taking, 25:56 it takes about 72 hours for that 25:59 first dose to reach its peak 26:04 at that dose. 26:06 If you give a two x dose or a three x dose, 26:10 you get a higher immune response each time, taking about 72 hours. 26:15 If you didn't use any more product, you slowly over 26:20 roughly a ten day to two week period of time, go back down to baseline. 26:24 Now that's a B in response. 26:27 Trained immunity is additional to immune response. 26:32 Again, this 72 hour take is based on 26:36 maybe somebody never having taken the product before. 26:40 Once you get into the trained immunity 26:43 track where you've got cells that will be coming 26:47 right out of the bone marrow, you're not looking at a 72 26:51 hour and you're not looking at a two week dwindle. 26:56 You're looking at something that comes right out of the gate, 26:59 right at the position that you have been operating on. 27:03 And let's say you 3 or 4 months down the road, 27:10 you have a heating bill 27:12 and you just can't afford the dose that you've been using. 27:17 Fine. 27:18 Don't cut the dose in half. 27:21 Take that same dose every other day. 27:25 That's the advantage that trim are trained to be. 27:28 It's going to keep you up there on top of the roof, even though you're 27:33 using the product every other day, effectively cutting your cost in half. 27:39 You want to have as many face cards in your deck as you can 27:45 for those that can relate to gambling. 27:49 And so when we're looking at cancer and we know cancer is evolving, 27:53 we know that they are 27:54 continually trying to figure out how to evade the immune system. 27:58 So they're there. They have changes. 28:01 You have sub clones. 28:02 You have all these different aspects in regards to cancer. 28:05 And so it becomes an important 28:09 to have this innate immune system to continually be trained, 28:14 you know, so that when they recognize a new version, 28:18 there are now new white blood cells are produced in the bone marrow. 28:23 And when they're coming right out of the gate, 28:25 they remember all the kind of past sub clones and past, 28:29 and that continually happens so that the immune system doesn't 28:33 stay on top of essentially the evolution of cancer. 28:37 And that's why you're saying making the point 28:40 that, you know, taking it for months. 28:42 That's great. 28:43 And but let's say financially it is difficult. 28:46 And obviously if that is the case, then do it every other day 28:50 so that you can still be on top of whatever 28:54 changes in adjustments that, that the cancer may, may be doing. 28:58 That's why it's so important to continually have. 29:01 I mean, it's like you, 29:02 you don't have just a police force to go out there 29:04 and then clean up the criminals, and then you send all the police force home. 29:08 You want to stay on top of things and ask things change and how things evolve. 29:13 You want to be there, ready? 29:15 If I've got 29:16 a military force this designed to protect me, 29:20 and then in the case of an attack, 29:22 kill things and blow things up you right down to the nitty gritty. 29:26 That's what we're talking about there. 29:29 We don't want them sleeping in the foxhole. 29:32 We don't want them playing, you know, with their neighbors 29:37 and picking their nose while 29:40 the enemy is creeping up on us. 29:42 We want to make sure that they remain 29:46 trained, razor sharp, 29:50 but we don't want them out there cutting things. 29:52 If there's no target. 29:54 Now, don't again, don't don't confuse training 30:00 of our innate immune system 30:03 with the acquired side of immune system 30:06 work, which is specific, again, the intelligence officers 30:11 that are looking for a particular antigen, 30:17 they're going to tag it with a specific 30:19 antibody complement this. 30:22 Another thing. 30:23 Anybody's tagged something. Guess what. 30:25 Here comes compliment. 30:27 And vice versa. 30:29 If complement tags something here comes antibody. So 30:36 each one 30:37 is a unique and specific system. 30:42 Whereas with innate immunity again 30:45 you're either non-self or self. 30:49 If you're non-self I'm going to attack 30:53 I'm going to eliminate and go back to it. 30:58 Rest with training our trim I'm going to do it faster. 31:02 That's the specificity 31:06 non specificity. 31:08 But some of this Bruce Lee stuff okay. 31:13 And you can mention in regards to because there are many different glucans. 31:18 And you talked about the different structures. 31:21 And we have glucans from, from different substances like the mushrooms. 31:25 And then you have, you know, so and they have different 31:29 quality and different impact on the system. 31:33 So the, the this 31:36 beta glucan is the one that is, is doing 31:40 has the best impact of the strongest impact in regards 31:44 to doing this kind of trained innate immune system that initiating the trim 31:49 in addition to like you're talking about the KR, you know, 31:52 the KR three, the complement receptor where you kind of tagging and then, 31:56 you know, the adaptive part of the immune system kicks in 32:00 with a material called k creston. 32:04 Now K creston is a mushroom sauce 32:10 made a glucan also 1316 in structure 32:15 but have a peer reviewed. 32:17 We're talking about a pharmaceutical here a true pharmaceutical. 32:21 It's only available with prescription. 32:26 Nobody in America 32:28 is selling a glucan from mushroom 32:32 that is equivalent to PSA crest. 32:35 And I know this because much of what I've done, particularly over 32:39 the last 20 years, is have universities test side by side 32:45 hundreds and hundreds of different materials, 32:47 mushrooms being part of that. 32:50 It's not that the mushrooms have made or 16 times the dose. 32:55 Can you drink enough water to swallow it? 32:57 No, but don't let that stand in the way of millions. 33:01 In fact, some cases tens of millions of dollars of sales 33:05 because people are easily persuaded by these slick brochures. 33:11 And it's not really fair in my mind, 33:14 to use pesky Creston studies 33:18 to talk about my product. 33:21 It's I don't mind either, because I think at the end of the day, we'll have helped 33:25 a lot of people. 33:28 And you talk to 33:29 just want to kind of expand a little bit again. 33:32 On you talked about the the PD one or Pd-l1. 33:35 Can I checkpoint inhibitors, you know that the tumor cells use 33:40 so that the immune system can't recognize them. 33:43 And, you know, just going to highlight that studies actually showing, 33:46 you know, the beta glue can you know, the type of beta glucan 33:49 you're talking about actually enhances 33:53 the immune system's ability then to see these cancers. 33:56 So if you bring in like immunotherapy, 33:59 beta glue can actually enhance the impact of that and has the effect. 34:03 So the immune system has a chance. 34:05 And to recognize that that cell and go after it and kill it. 34:10 So it is phenomenal how such a substance 34:13 can have such an impact on the muse system. 34:16 It's almost like like you're saying when you're picking teams 34:21 that our immune system evolve with knowing that this exists 34:26 and that this is something that we can benefit from to real enhance 34:31 our immune, you know, immune function in fight against cancer, 34:35 in the fight against infections and fight against whatever that may be. 34:40 If we did not have the relationship that I described, 34:44 where I said the immune system's constantly adapting, overcoming whatever 34:50 challenge, whether it's radiation, whether it's toxicity, 34:54 whether it's plastics, whatever it is, our immune systems. 35:00 But we we as individuals, we've got to participate. 35:03 We can't just get up and pray and nothing against praying. 35:08 I'm all about it to cover all the bases, but we need 35:13 it doesn't hurt though, and I'll quote doctor. 35:17 Doctor, I love that because he is arguably not only one of the top 35:22 immunologists in the world, but particularly in this arena 35:25 of how the immune system responds to glucans. 35:29 As a family, there's no telling how many thousands 35:32 of comparative evaluations that he's done over 30 plus years. 35:38 But he has said 35:41 that if 35:41 you want your immune system to recognize 35:44 and kill more cancer cells, 35:47 you'll use a little bit of this product each and every day. 35:51 And I tell him, I said, doctor, I'm 100% with you, 35:55 but the FDA will not let me put that on the side of the bottle. 36:01 I might have to, you know, get Doctor Karlfeldt 36:03 and his colleagues understanding these things. 36:07 Then they can say it. 36:09 And you do. 36:10 I know you do a great job of helping 36:12 your patients, their families and so forth, but it's so simple. 36:16 And I've said it before, I'll say it again. 36:18 The immune system and genetics, whatever 36:21 the answer is that we one day be cancer free. 36:25 I will assure you that those things are going to be part of the puzzle. 36:30 Genetics. 36:32 We have learned a tremendous amount. 36:34 That's part of what we've learned with trim. 36:37 We've seen these thousands and thousands of genetic switches 36:41 being clicked on to be able to leave those post-it notes in certain places, 36:47 giving us better odds of beating cancer. 36:50 So there you go. 36:53 I love it. 36:54 Oh, it's I mean, you're such a wealth of information. 36:57 And thank you so much for being the voice in the desert, you know, 37:01 and to really kind of drive this message forward, you know, thank you so much. 37:06 I appreciate you. And thanks to all that. 37:08 Come and listen. 37:09 We hope that some pearl of wisdom stays with you to help yourself, 37:14 your family, your loved ones, your your colleagues, your patients, whoever. 37:18 And so cherish that pearl of wisdom and pass it along. 37:23 Okay. Thank you.
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