Using FSM To Reduce Insulin Resistance & Support Pancreas

Carolyn McMakin, MA, DC

Thank you very much. It is a pleasure to be here. This is one of my favorite topics.

Beverly Yates, ND

I am so glad.

Carolyn McMakin, MA, DC

I am glad you are doing this summit.

Beverly Yates, ND

Thank you. I am looking at your work and your contributions. I just want to thank you in front of our audience for being a leader, for thinking outside the box, and for being willing to put that clinical rigor and proof-based scientific evidence behind doing this. Sometimes, when people step out, they are not always rewarded or embraced. I just wanted to say thank you for that.

Carolyn McMakin, MA, DC

You are very welcome. It became pretty obvious pretty quickly that FSM was going to be effective when we started treating muscle and nerve pain. Then there are things like asthma, where, in order to survive, you have to be evidence-based in this world. I started by teaching and then by publishing. Thanks to Leon Chaitow, Elsevier published the textbook.  There were more publications that followed, including Controlled Trials out in Ireland, and then the Consumer Book had to follow. In medicine, you have to do things in a particular order. In order to even qualify for a blinded, double-blind, placebo-controlled trial, you have to have published case reports in order to even get past some that are being considered. The important thing for me is that I was a pharmaceutical salesman for 16 years and understand how medicine and the FDA work. The important thing for me was surviving after I was gone. In order to do that, you have to be willing to put in the work. It has been a labor of love, and it has been really rewarding to watch practitioners get the same results with patients. It is reproducible for those effects. That is the important part.

Beverly Yates, ND

Absolutely. Having it be reproducible and helping people in the real world is always what we are after. In our talk, we are going to go over your use and development of frequency-specific microcurrent to reduce insulin and leptin resistance and provide support. One of the key organs, certainly when we are talking about diabetes, is the pancreas.

Carolyn McMakin, MA, DC

Okay.

Beverly Yates, ND

When you are ready, you are welcome to just share your screen, and we will record your slides and your presentation.

Carolyn McMakin, MA, DC

Okay. Sounds good. FSM can help reverse type 2 diabetes, I think. First, FDA compliance: you cannot make any claims about being able to diagnose, mitigate, treat, or prevent any condition or disease. But I can present data that supports our theoretical ability to be useful. I am just a clinician sharing my clinical experience with your clinicians. The devices we use are approved by the FDA in the category of TENS devices for the treatment of pain. Even though what you are treating is insulin resistance or type 2 diabetes, you have to have a pain diagnosis. Anyone knows that an insulin-resistant patient with a 45-inch waistline who does not have low back pain. We have evidence that we can reduce blood sugar levels and improve insulin resistance. I have no studies that show that we can reverse type 2 diabetes. But the theory is there, and that is what we are going with in this presentation. I do teach seminars and frequency-specific microcurrent, so there is a conflict of interest statement, but I really have an interest because I treat myself every night. I am 76 years old, and it is really important not to be a Type 2 diabetic.

What is FSM? Frequency-specific therapies were developed in the early 1900s by MDs and naturopaths in the U.S., U.K., and Germany, and they were used by thousands of physicians until 1934 because of the Flexner Report. Medicine labeled them all as ineffective fakes. Drugs and surgery were to be the only tools of medicine. Nutrition, herbs, homeopathy, and frequency therapies were all outlawed, and every medical intervention except prescription medication was outlawed. Any physician who used these tools would lose his license. The devices went in the back rooms of clinics. All the research in history was lost. How did somebody decide in 1920 that nine hertz would address the pancreas and 91 hertz would address the eyelids? It is gone. When you cleaned out your grandfather’s library after he passed away, that was it. Electronic copies of the Electro Medical Digest ended up in the rare book room at the Naturopathic College. Some practitioners who did not succumb were persecuted and put in jail.

FSM came about because of Harry van Gelder. This gentleman was an osteopath and naturopath from Australia and the U.K. He bought a practice in 1946 and walked into the back room of the clinic and found a device that probably looked something like this under a sheet. That device was made in 1922, and it came with a list of frequencies. In 1983, my husband and partner, George Douglas, worked with van Gelder and brought home a copy of the frequency list and put it in a drawer. The list looked like this: It was just numbers, and next to the numbers were words. We assumed that 40 hertz would neutralize inflammation, not cause it. We assumed that 22 hertz would address the small intestine and 97 hertz would address the adipose.

I met George in 1991. I finished Chiropractic College in 1993, and when we moved into my new clinic in 1995, George was moving, and he found the frequency list in a drawer. We had a precision micro with two channels, and he said we could use Harry’s frequencies on this microcurrent machine and see if it worked. It turns out that it did. But we have to live with the fact that information about how the frequencies were derived is lost. We are never going to find out. The mechanism of action is hypothesized but not proven. In the 1920s, equipment was not microcurrent, and the 1920s therapy was not FSM. That is our history. We use two channels at one time that cross through a field, and in that field in the middle, there is the frequency from Channel A and the frequency from Channel B. Then you get the sum of the two channels and the difference between the two channels. We are not sure which portion of the field produces the effects, but we do know from experience. It literally took 50,000 patient visits over five years to convince me that the frequencies always did what they were alleged to do.

We used them first in 1995. I taught him in 1997 to find out if the results were reproducible because we were doing things in nerve pain, muscle pain, and scar tissue that were just impossible, and it turned out in June 1997 that the results were not only teachable but reproducible, and then research on animals and humans and clinical results have accumulated over 30 years now. We have 20 peer-reviewed papers, a medical textbook, and a similar book, and there are now 5,000 FSM practitioners in 23 countries. The blinded animal research we did at the University of Sydney in Australia showed a 62% reduction in white box Sudanese-mediated inflammation. They painted arachidonic acid on the mouse’s ears, they measured the mouse’s ears, and then they did something to the mouse—they gave it a drug—or they did something before or after the arachidonic acid.

We had originally seen a 70% reduction in LOX-mediated inflammation, and the researcher called a halt, sent everybody home, brought everybody back the next day, and blinded everybody, but not literally, but put them in a room with the windows covered and everybody being careful. She put in a placebo frequency. We had a 62% reduction in LOX-mediated inflammation. Lipoxygenase is one of the prostaglandins, the mandatory prostaglandins, and she had never tested any drug, prescription or non-prescription, or reduced lipoxygenase by more than 45% in 20 years with the research. She had a 30% reduction in LOX-mediated inflammation, which does not sound good, but it is equivalent to injectable Toradol. When she tested it on the same animal model, all the animals responded. There were no exceptions, and it turned out to be a four-minute time-dependent response. Half the responses were present for 2 minutes. Half the responses were present for 4 minutes. Then she tested three others and four others of our frequencies, including one that reduced inflammation by 40 hertz in the skin, which is a tissue on the mouse’s ears, and only 40 hertz on Channel 8 and 116 hertz on Channel B reduced inflammation. Then we had a treatment protocol for fibromyalgia associated with spine trauma. 

I presented 27 cases at the NIH. By the time we published the paper, there were 54 patients, and we had blood sample data from the NIH. Six of them with a control patient had just myofascial trigger points, not full-body fibromyalgia. This is what the pain diagram looks like. Fibromyalgia is definitely one of the more difficult conditions to treat. We found that only one frequency combination that reduced pain had to be rung from the patient’s neck to the patient’s feet: polarized, positive, current. They came in with their pain, an average of 7.4 and went to 1.3 in 60 minutes. The relief lasted from 2 hours to 2 weeks. All patients had pain relief, and 58% recovered within four months. Recovery was individualized; of course, we had FSM in the office. Some patients needed a home unit to keep their pain below four during physical therapy reconditioning, and some of them took supplements. 13 of 54 patients discontinued treatment for reasons not related to treatment side effects. The biggest challenge is actually getting patients used to dropping pain.

Then, in diabetes, it is going to be getting them used to a change in diet—what they considered comfort foods. This is what happened to the inflammation in that group. Interleukin 1 went down from an average of 330 down to 80, plus or minus. Look at the P-value with only six patients. to do that, no statistic sticks. A P-value of 004 is huge, and the other P-value is on time points to get this kind of reduction in 90 minutes. The P-value is actually three zeros and one interleukin 6 to 0 and an eight. A huge statistical significance in a small group, ten of alpha, two zeros, and two. CGRP is involved in insulin resistance, which we know about. That went down. It was a vasodilator and neurogenic inflammation that went down. You notice that they all stop in the normal range. That is important because you do not want to take inflammation down too far or for too long. Before we get into the diabetes stuff, I just need people to think about how is it that frequencies and microcurrents can do what we just showed that it did.

Newtonian physics describes large objects, but it falls apart at the molecular level. Your body is a large object made of molecules, atoms, and subatomic particles. Your body is theoretically a quantum object held together by electromagnetic bonds. In physics, every bond has a frequency at which it resonates, including, let us say, insulin, fascia, and every connective tissue. How does the body conduct current? Your body is a semiconductor, so copper is a conductor, and ceramic is an insulator. In your body, 85% is water. Where does the water live? It lives in the blood, and it lines the gel inside the cells. We have the idea that the cells are just fluid-filled sacs. Well, they are not filled with fluid. They are filled with a gel matrix that holds all the organelles in position. This matrix and the blood have structured water that forms a structure that is much like silicon, with a space and a predictable place for an electron to flow. The flow of current inside the cell and outside the cell in the blood is very much like silicon. Your body is a big computer chip. Water lines to go inside the cells and start forming structures that act as semiconductors. A biophysicist named Saint George published this in 1986. Your body is an electromagnetic system that looks solid, but the cells function as a semiconductor network that conveys charge and information in the form of signaling that is mediated by frequencies.

What is the big deal with resonance? Resonance is the tendency of a system or a bond to oscillate or vibrate at large amplitudes in response to some frequencies and not others at the resonant frequency. Very small forces can produce very large-amplitude vibrations. They found in the 17th and 18th centuries that soldiers marching in step could create a vibration that would collapse a bridge. That is a huge vibration made by just men walking in steps. To this day, a company of soldiers walking across the bridge breaks step, and then they go back and stop when they go across. What is resonance? Two tuning forks tuned to the same frequency will vibrate in resonance with each other from across the room. When one tuning fork is struck. Resonance took down the Tacoma Narrows Bridge. It was not a hurricane. It was a simple northwest storm, but the bridge was flexible. When the swaying of the bridge reached the resonant frequency of the bridge, the thing, the sine wave, just came apart. Biological resonance explains the effects on living tissue. We are used to drugs and even supplements that act like keys in a lock to change membrane receptors and intracellular function.

You have to look at how a cell actually does its business. The outside of the cell is filled with protein receptors, and those receptors are sensitive to outside inflammation. The circulation has pathogen-associated molecular patterns and damage-associated molecular patterns. They hit that receptor that signals the kinase, which signals the transcription factors, which signals gene expression, and that is what makes the cell. Any cell in your body can turn on the genes that create an inflammatory cytokine. You take ibuprofen, and it acts like a piano lock to change this receptor. Frequencies appear to change that configuration electromagnetically, like the key fob on your car. You can now open your car from across the parking lot, and your key remote sends a signal. Exactly, and only to one car. There are 12 blue Subarus in a row, and your key fob opens only yours. Ever since, it seems to work with specific problems in specific cells.

Let us look at type 2 diabetes. Type 2 diabetes occurs when the insulin-producing beta cells of the pancreatic islets are unable to produce or release sufficient insulin to overcome peripheral insulin resistance, resulting in hypoglycemia. Basically, type 2 diabetes starts with a virus or some infection, but the general thinking about type 2 diabetes is that you are insulin resistant, and the pancreas pumps out more and more insulin to try and overcome the insulin resistance in the peripheral tissue. The pancreas just wears out. It just gives up. There is a lot of complicated chemistry in here that you all know about.

That is not my specialty. My specialty is changing insulin resistance. Why do you want to reduce insulin resistance? Well, the metabolic effects are obvious. immune-compromised inflammation. Insulin resistance is one of the major causes of Parkinson’s amyloid plaques and white matter injury in the brain that lead to Alzheimer’s, vascular cognitive impairment, and vascular cognitive impairment. All of the late-age cognitive deficits are related, starting with the inflammation that comes from insulin resistance, vascular effects, and the theory of dysfunction. Arterial stiffness is a constriction in hypertension. Insulin resistance is one of the major causes of heart disease. There are a lot of reasons to reverse type 2 diabetes, reduce insulin resistance, avoid Alzheimer’s, heart disease and stroke, kidney failure, and macular degeneration. There is nothing good about insulin resistance. I am going to go through this because this is your specialty.

The pathogenesis of hypoglycemia and type 2 diabetes starts with insulin resistance. If you can reduce that improved muscle tone to increase glucose uptake and treat the pancreas, you do not necessarily need to produce more insulin because you already have it. You need to repair the pancreas in a perfect world. The two main factors that contribute to this resistance are excess body fat, especially around the belly. The fact of the matter is that when you are insulin resistant, the body puts excess adipose tissue around it because of a lack of physical exercise. What really causes insulin resistance? There are slides that your team can look through. Endoplasmic reticulum stress occurs when the capacity of the reticulum to fold proteins becomes saturated, and that can be caused by factors that impair protein glycosylation, which means the sugar cannot be put in the muscles. Mitochondria dysfunction: I think your team knows all of this. The liver produces sugar from glycogen, and this becomes important when we start talking about treating the vagus nerve. Here is TNF alpha in the adipose and interleukin one. TNF-alpha interleukin one: I have data that says that we can reduce interleukin 1 and 2 in alpha. There are some more, and your team knows about this. These slides will be available to them.

In our world, there was an internist who created what we have called the insulin resistance protocol since 2003, and he gave us this protocol. treating the adipose for toxicity, inflammation, chronic inflammation, trauma, and emotional factors and just getting it to work better, he felt that a virus was involved in insulin resistance, and that has been proven since then, as well as a nonspecific infection that he identified. 97 is the frequency for the adipose. When I first brought this protocol home in 2003, I used it on my staff and myself, and it caused everybody to reduce their appetite in general, especially for sweets, reduce their waist size, and become constipated. It caused constipation in every patient treated. This naturopath, the first time we treated him, had his staff there, and he found out that if he treated toxicity in the parasympathetics, there was toxicity in the adipose, and the toxicity went into the parasympathetics. When we did that, it reversed constipation. We have had the insulin resistance protocol since 2003. Recently, it became obvious: What controls insulin? Well leptin. What is the point of treating insulin resistance unless you treat exactly what causes it? selective insulin and leptin resistance, and metabolic disorders. 

This is the paper where your people can look it up: Cell Metabolism. Volume 16 was published in August 2012. There is a diagram of the pattern where leptin controls insulin, leptin causes resistance, leptin is released from fat cells, and leptons are hormones and cytokines. Reducing inflammation in the adipose tissue should reduce leptons. Leptons are secreted by adipose to control insulin. Leptin and insulin directly regulate each other, and leptin inhibits insulin. This pattern of insulin stimulation stimulates leptin synthesis and secretion. As the pancreas produces more insulin to counteract insulin resistance, leptin goes up. It is a bad feedback loop between leptin therapy, insulin sensitivity, and glucose homeostasis. This paper was also published in 2012, so you can look that reference up. Leptin increases insulin sensitivity, so leptin resistance increases insulin resistance. There is that paper that was published in 2008: Inflammation, interleukin-6, and serum increase leptin resistance. 

This whole research-based concept makes your brain hurt. But leptin and insulin enter the brain from the plasma. Leptin and insulin resistance are related to inflammation and stress, so the handle that FSM has is our proven ability to reduce inflammation. This is all related to interleukin 6 and interleukin 1. Increased stress increases cortisol, and increased cortisol reduces melatonin. Low levels of melatonin lead to leptin resistance. Low levels of melatonin lead to leptin resistance. Melatonin is made by the pineal gland, and while cortisol decreases melanin, melatonin increases it. If you can decrease stress and cortisol, melatonin will go up. That leads to increased leptin sensitivity, which leads to insulin sensitivity. There is a paper published in 2005 on the relationship between melatonin and cortisol, limit rhythms, leptin, resistance, stress, and melatonin. 

There is a point to all this. Hang in there. Increased stress increases cortisol; increased cortisol reduces melatonin; low levels of melatonin lead to leptin resistance; stress increases cortisol that decreases melatonin; higher cortisol and lowered melatonin lead to leptin resistance; and leptin resistance leads to insulin resistance. It turns out that the way to reduce leptin resistance and insulin resistance is to decrease inflammation, decrease cortisol, and decrease stress. We have data, thanks to Roger Callahan. He did a study on heart rate variability. This was after the launch. The parasympathetic levels were high because it was after launch, which is a pretty sympathetically driven M.D. with a busy practice. He was medical director of NASA for ten years, and he ran the frequencies to quiet the parasympathetic. Then he waited for 2 minutes, and then he retested. This is the 62nd treatment, and the parasympathetic response has dropped by 50%. Then he increased the sympathetic, and the parasympathetic pretty much disappeared. One-minute treatment. This is 2 minutes, one minute each, and a two-minute wait to make sure you are measuring what is happening in the system instead of the frequencies, and then he increased secretions; that is what 81 is for: increased secretions of the parasympathetic vitality, not very sympathetic. Everything returned to normal. We have data that shows that frequencies can reduce stress and return to parasympathetic balance. So leptin, insulin, cortisol, and melatonin. Cortisol should drop at night, and melatonin should increase with insulin. Since leptin sensitivity should increase. Okay, so this is a circadian rhythm. You are all familiar with that.

With FSM, we created a protocol that you treat, observe, and then create based on the research. We have always had, since 2003, the insulin resistance protocol. Leptin resistance was created based on research to reduce inflammation in the adipose, that is a given. Then quiet the parasympathetic. That research shows that we could reduce cortisol by quieting the adrenals, and I have more on that that I have not put up here, but we could reduce cortisol as demonstrated in 2001 and then treat the pineal gland. Now this is theoretical, but this is to increase secretions in the pineal gland. But then the patient has to cooperate with good sleep hygiene, reduce light, relax, and not eat light. But the vagus in all of this is the missing piece. The vagus nerve suppresses and controls the immune system and inflammation. The vagus nerve suppresses T-cells and macrophages, and it suppresses the conversion of liver glycogen into blood sugar. The vagus nerve is suppressed by infection, stress, and trauma. The key to insulin and leptin is to keep resistance low, reduce inflammation, and keep the vagus nerve turned on.

Elevated cortisol and sympathetic neurotransmitters tell the brain there is stress. 

I run this protocol on myself every night, and people ask why. I said, Well, did you watch the news? Did you drive on the freeway? Of course, there is stress. Elevated insulin tells the brain there is stress. Elevated leptin tells the brain there is stress. The brain suppresses the vagus nerve because it is suppressed by infection, stress, and trauma. The vagus nerve reduces sympathetic tone. The vagus nerve reduces inflammation, and the vagus suppresses glucagon, which is converted into sugar by the liver. When the sugar is high, the pancreas has to work super hard to produce enough insulin to drive it down. We use FSM to treat the vagus by quieting the midbrain. We know this works because we are able to reduce limbic pain and stress by reducing the activity of the limbic system, the cortex, which senses stress, and the medulla, which is the source of the vagus nerve. Then 40 and 116 were the frequencies; the reduced LOX and COX in mice reduce inflammation directly and then turn on the vagus to reduce trauma, increased secretions, and vitality in the vagus. We know that it works because we have been able to briefly reverse ventricular tachycardia and stop atrial fibrillation by increasing secretions in the vagus, but that has not been published yet.

What turns down the vagus infection is stress and trauma. A cold infection, if you take it three times, will not show up on an x-ray. A hidden infection activates the immune system, causes increased inflammation, reduces vagal tone, and increases the conversion of glycogen stored in the liver into sugar to combat those perceived threats by the immune system. The infections we deal with are root canals, mold, maybe chronic lyme, etc. 

What are the main stressors? Sleep deprivation: the idea that people think they can get by on 4 to 5 hours of sleep a night—I do not know where that came from, but that is not according to the literature. Work habits or requirements: long hours on the job, alternating day and night shifts, or an inherently stressful job. Police officers, for example, have personal habits. I do not go for a walk. Interpersonal conflict on the job, at home, or in interpersonal relationships: sleep apnea is a huge stressor. Sympathetic stress created by sleep apnea causes nighttime conversion of stored glycogen into sugar to increase, sympathetic tone to increase, blood pressure, risk of heart attack, and risk of stroke to increase, and daytime and nighttime sugar to increase by increasing sympathetic tone. We never think of testing for sleep apnea when it comes to type 2 diabetes, and it is a primary driver. In-home sleep studies are less expensive and more reliable. I use the Watch app, but there may be others. The Watch app is the one I use for education, encouragement, and support for the CPAP or appliance use. 

I have patients and practitioners who are just terrified of the CPAP, and I have been using one for eight years, and it has made a huge difference. Sleep apnea is huge. There is a book called The Promise of Sleep by William Dement that everybody should read. It is huge. Insulin resistance and leptin resistance: here is the insulin resistance protocol: first we treat leptin resistance, then we treat the vagus reduced-spectrum inflammation, leptin resistance, and insulin resistance. But there is also diet, excess calories, carbohydrates, and fat that get converted into sugar, exercise, and reduced muscle tone. You can ask a patient, Do you have 10 minutes at night? 10 minutes to go for a walk is a reasonable thing to do. If you tell them to go for a walk for an hour, they are not going to do it. But you ask them to change one thing, and that is to go for a walk 10 minutes after dinner. Genetics, you cannot change; habits, you can change; depression and anxiety can go down when you treat leptin, insulin resistance, and inflammation in the brain because they contribute to depression and anxiety. It is collaborative. You and your patient form a team to educate and motivate them. If you want to avoid Alzheimer’s and early cognitive decline, you really need to take this seriously. They need to understand the contributing factors of lifestyle and diet risks and the long-term consequences if they do not educate themselves.

I had one practitioner say it is easier to modify somebody’s religion than it is to modify their diet. But I have to eat French fries. No. Or how about I have to eat white rice? Well, how about wild rice? Nutritional support for blood sugar control—your naturopaths have a handle on that. Berberine is the one that my doctor uses for enjoyable, consistent, mild-to-moderate exercise. They do not need to run. They can walk. Then FSM is a helpful adjunct to reduce inflammation, insulin, and leptin resistance, and improve vagal tone. Things that are impossible to do any other way, at least not in 4 minutes. You need to convince the patient that reversing type 2 diabetes is worth the effort because it requires a huge change. 

The Resonance Effect is the book that North Atlantic Books, Penguin, and Random House treated. You can get it on our website at frequencyspecific.com. It is a page-turner and really takes about 4 hours to read. It is pretty fun, and then there is the fact that I teach a personal seminar, so that is my conflict of interest, but we do it in five days. I used to do it in three, then three and a half, and I finally gave up because I did not know what to send them home without. It all includes 4 hours and 10 hours of hands-on practicum. If you take the video training, you get 4 hours of individual training to learn how to use it. You are overwhelmed at the end of the five days, but you are saved. You are not going to hurt anyone. three months up to a forever learning curve. In the last 12 months, I have used frequencies I have never used before. In our 30 years of practice, we have had advanced courses, webinars, and workshops. Our equipment is inexpensive compared to a lot of the stuff that you buy. It is U.S. aid. It is not a Chinese knockoff. No offense to those who have those 510K-approved ISO certifications. It has a CE mark for those of you practicing in Europe. It is possible that it is easier with that person, and it is definitely worth the effort and makes it easier, faster, and more efficient. Then our seminars are listed at frequencyspecific.com. You can also start with the book. There you go.

Beverly Yates, ND

That was wonderful.

Carolyn McMakin, MA, DC

Does that make sense?

Beverly Yates, ND

Yes, it certainly made sense to me.

Carolyn McMakin, MA, DC

But I skipped over a lot of the parts that you are experts in, so I will let you cover those.

Beverly Yates, ND

Yes. Thank you. Throughout the summit, we will cover the things that you skipped. I knew. I knew. I skipped them as we covered it elsewhere. But the things that were not repeated elsewhere are what you covered and dove into.  Thank you so much, Dr. Carolyn McMakin, for making that clear and for a great journey through the way to use frequency-specific microcurrent to help people quickly make a difference in a really thorny, complex web of things that are wrong behind the scenes that make such a difference for reversing Type 2 diabetes and pre-diabetes and achieving that blood sugar control and being able to maintain it.  I think when a lot of people are told what I call the old 1950s advice that no longer applies of eating less and moving more, and you look behind the scenes and listen to your entire presentation, you realize, Oh, that is why eating less and moving more is inadequate. In today’s busy world, what I sometimes call Cortisol Nation.

Carolyn McMakin, MA, DC

Yes, that is a good word for it. Part of the challenge with patients is that you tell them in a year they are going to feel better for it, and we do not have the patience. Patient compliance is the biggest problem you have in reducing type 2 diabetes. Change the diet, fine. Do you have 10 minutes after dinner? Well, yes. I sit down and watch television, but instead, put down your fork and knife, put the plate in the sink, grab your dog, and go out the front door and walk for 5 minutes out, 5 minutes back. You can do that. Well, and then at their next visit in two weeks, you have them walk 15 minutes out and 15 minutes back. But if they do not see results in the first month or two, you are going to lose them. They are not going to stick with it for a year. However, it helps you get that done faster.

Beverly Yates, ND

Absolutely. I think bringing up the issue of people’s ability to cooperate with a treatment plan and willingness to hang in there is important, and often we are taught, certainly here in the U.S., that we should expect instantaneous results. One pill will fix things in the next 30 minutes when we have actually spent years and decades in a destruction cycle, perhaps not even understanding how much our health is being undermined, or maybe we are making choices that really do not support us. In that process, it is such a tragedy because there is so much harm that people are experiencing that is completely unnecessary. Some cultures have that walk after a dinner break. Did they have a saying for it? I think in Italian it is Passeggiata. I probably did not say that. It is in the Chinese culture; it is in a variety of cultures, and you just go, Wow, we need to be doing that globally. Go for a walk after dinner.

Carolyn McMakin, MA, DC

Those cultures have a body mass index that is so much lower than ours. They just do not understand why Americans are so fat. Yes. It is a habit. It is the things that they eat. It is how they deal with toxins. They do not use glyphosate in Italy. It is not allowed in France; it is not allowed in Germany; it is not allowed. So you are in a less toxic environment, you go for walks, and you eat differently. The stressors are different. I mean, the U.S., as you said, is just a stressed nation.

Beverly Yates, ND

It does not serve us. It is a large part of the puzzle. Not everything can be quantified in a test tube. But sometimes the answers are right in front of us. We really have to shift how we live and take control of it. One of the things my family did back in the early 2000s was add a spinning bike to our living room. that way if we are listening to music, reading books, watching TV, or just whatever. If there is not good weather, and I am fortunate to live here in California, it is usually wonderful weather, but on the occasions when we do not have it after dinner, just stick it on. It has been, just, just do it. Get moving,

Carolyn McMakin, MA, DC

That is the way. 

Beverly Yates, ND

Yes. Thank you so much, Dr. McMakin, for sharing your insights, your experience, and your brilliance with our audience. I am really glad to have you as part of the summit. I love being able to invite people who are pioneers, who are thinking outside of the box, and who have tangible, proven ways to support people, particularly for things that they might not otherwise know about. also share that with clinicians. 

Our audience is comprised of the general public as well as health and medical professionals. Your talk here is probably going to be at the level of the health and medical worlds. But for anyone who watches and is in the general audience, please just take notes and really think about what Dr. McMakin shared with us, because these are parts of the puzzle. You saw all those various hormones. You saw leptin, cortisol, and insulin being talked about. There are all these players that affect blood sugar regulation. So if you are watching this and you have really struggled with your blood sugar regulation and you feel you have taken care of the basics, I invite you to check out if this is something that might be supportive for you.

Carolyn McMakin, MA, DC

It makes it happen faster, is less expensive, helps speed up the process, and makes everything you do more effective. It is a real privilege to be able to contribute to the health of the nation, and I thank you for what you have done to make this known. Yes, you too. Thanks for having me. I appreciate it.

Beverly Yates, ND

Absolutely. Friends, please check out what Dr. McMakin is sharing. Would you tell us where they can connect with you and your work? I know you showed it in your slide, but I want to be sure we get this for the audio.

Carolyn McMakin, MA, DC

Frequency-specific.com on the website. A good place to start is the resonance effect. It tells the story of FSM and how it developed because otherwise it is hard to believe that now where we ended up after 30 years, when you read the story of how it started and it ends patient case reports, you can see how it was developed, how it works, and how it can help you recover. Patients can find a practitioner at frequency-specific.com. There is a need to find a practitioner, and the devices have to be prescribed by a practitioner. There is a way that we can help.

Beverly Yates, ND

Great. It sounds like quality control is baked in, which is so important. I understand why that matters to you.

Carolyn McMakin, MA, DC

Yes.

Beverly Yates, ND

Great. Thank you so much.

Carolyn McMakin, MA, DC

Good bye.